β1-Adrenoceptor, but not β2-adrenoceptor, subtype regulates heart rate in type 2 diabetic ratsin vivo
Author:
Affiliation:
1. Department of Physiology, Otago School of Medical Sciences, HeartOtago; University of Otago; Dunedin New Zealand
2. Department of Physiology, Faculty of Medical and Health Sciences; University of Auckland; Auckland New Zealand
Funder
University of Otago
Otago Medical Research Foundation
National Heart Foundation of New Zealand
HealthCare Otago Charitable Trust
Publisher
Wiley
Subject
Physiology,Physiology (medical),Nutrition and Dietetics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/EP086293/fullpdf
Reference40 articles.
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2. Altered contractile response due to increased β3-adrenoceptor stimulation in diabetic cardiomyopathy: the role of nitric oxide synthase 1-derived nitric oxide;Amour;Anesthesiology,2007
3. Localization of f-channels to caveolae mediates specific β2-adrenergic receptor modulation of rate in sinoatrial myocytes;Barbuti;J Mol Cell Cardiol,2007
4. Chronic diabetes alters function and expression of ryanodine receptor calcium-release channels in rat hearts;Bidasee;Mol Cell Biochem,2003
5. Decreased catecholamine sensitivity and β-adrenergic-receptor density in failing human hearts;Bristow;N Engl J Med,1982
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