Opposing modulation of Cx26 gap junctions and hemichannels by CO 2
Author:
Affiliation:
1. School of Life Sciences University of Warwick Coventry UK
2. School of Chemical Engineering and Analytical Science University of Manchester Manchester UK
3. Department of Biosciences Durham University Durham UK
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP280747
Reference60 articles.
1. Gap junctional intercellular communication capacity by gap-FRAP technique: A comparative study
2. Conformational changes and channel gating induced by CO2 binding to Connexin26;Brotherton DH;bioRxiv,2020
3. Deafness induced by Connexin 26 (GJB2) deficiency is not determined by endocochlear potential (EP) reduction but is associated with cochlear developmental disorders
4. Cx26 keratitis ichthyosis deafness syndrome mutations trigger alternative splicing of Cx26 to prevent expression and cause toxicity in vitro
5. Elevated Carbon Dioxide Blunts Mammalian cAMP Signaling Dependent on Inositol 1,4,5-Triphosphate Receptor-mediated Ca2+ Release
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1. Structures of wild-type and a constitutively closed mutant of connexin26 shed light on channel regulation by CO2;eLife;2024-06-03
2. Structures of wild-type and a constitutively closed mutant of connexin26 shed light on channel regulation by CO2;eLife;2024-06-03
3. Structures of wild-type and a constitutively closed mutant of connexin26 shed light on channel regulation by CO2;2024-04-16
4. Structures of wild-type and a constitutively closed mutant of connexin26 shed light on channel regulation by CO2;2024-01-08
5. X-linked Charcot Marie Tooth mutations alter CO2 sensitivity of connexin32 hemichannels;Frontiers in Cellular Neuroscience;2023-12-21
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