Contribution of small conductance K+channels to sinoatrial node pacemaker activity: insights from atrial-specific Na+/Ca2+exchange knockout mice-Reference-Cited by-同舟云学术

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Contribution of small conductance K+channels to sinoatrial node pacemaker activity: insights from atrial-specific Na+/Ca2+exchange knockout mice

Published:2017-05-13 Issue:12 Volume:595 Page:3847-3865
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Torrente Angelo G.¹,Zhang Rui¹,Wang Heidi¹,Zaini Audrey¹,Kim Brian¹,Yue Xin¹,Philipson Kenneth D.²,Goldhaber Joshua I.¹³^ORCID

Affiliation:

1. Cedars-Sinai Heart Institute; 8700 Beverly Blvd Los Angeles CA 90048 USA

2. Department of Physiology; David Geffen School of Medicine at UCLA; 650 Charles Young Drive South Los Angeles CA 90095-1751 USA

3. Department of Biomedical Sciences; Cedars-Sinai Medical Center; 8700 Beverly Blvd Los Angeles CA 90048 USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1113/JP274249/fullpdf

Reference54 articles.

1. Apamin modulates electrophysiological characteristics of the pulmonary vein and the sinoatrial node;Chen;Eur J Clin Invest,2013

2. Distinct localization and modulation of Cav1.2 and Cav1.3 L-type Ca2+ channels in mouse sinoatrial node;Christel;J Physiol,2012

3. Evidence for a novel bursting mechanism in rodent trigeminal neurons;Del Negro;Biophys J,1998

4. Effects on atrial fibrillation in aged hypertensive rats by Ca2+-activated K+ channel inhibition;Diness;Hypertension,2011

5. Inhibition of small-conductance Ca2+-activated K+ channels terminates and protects against atrial fibrillation;Diness;Circ Arrhythm Electrophysiol,2010

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