Molecular mechanism for muscarinic M1 receptor-mediated endocytosis of TWIK-related acid-sensitive K+ 1 channels in rat adrenal medullary cells
Author:
Affiliation:
1. Department of Cell and Systems Physiology; University of Occupational and Environmental Health School of Medicine; Kitakyushu 807-8555 Japan
Funder
Salt Science Foundation
Japan Society for the Promotion of Science
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/JP275039/fullpdf
Reference56 articles.
1. Modifying the subunit composition of TASK channels alters the modulation of a leak conductance in cerebellar granule neurons;Aller;J Neurosci,2005
2. Emerging roles for two-pore-domain potassium channels and their potential therapeutic impact;Bayliss;Trends Pharmacol Sci,2008
3. Differential phospholipase C-dependent modulation of TASK and TREK two-pore domain K+ channels in rat thalamocortical relay neurons;Bista;J Physiol,2015
4. Selective inhibition of receptor-coupled phospholipase C-dependent processes in human platelets and polymorphonuclear neutrophils;Bleasdale;J Pharmacol Exp Ther,1990
5. Roles of G beta gamma in membrane recruitment and activation of p110 gamma/p101 phosphoinositide 3-kinase gamma;Brock;J Cell Biol,2003
Cited by 15 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Regulation of Src family kinases by muscarinic acetylcholine receptors in heterologous cells and neurons;Frontiers in Molecular Neuroscience;2024-01-11
2. Enhancement of muscarinic receptor–mediated excitation in spontaneously hypertensive rat adrenal medullary chromaffin cells;Autonomic Neuroscience;2023-09
3. Muscarinic Receptor Stimulation Does Not Inhibit Voltage-dependent Ca<sup>2+</sup> Channels in Rat Adrenal Medullary Chromaffin Cells;ACTA HISTOCHEMICA ET CYTOCHEMICA;2023-08-30
4. Adaptive remodeling of the stimulus-secretion coupling: Lessons from the ‘stressed’ adrenal medulla;Vitamins and Hormones;2023
5. PACAP and acetylcholine cause distinct Ca2+ signals and secretory responses in chromaffin cells;Journal of General Physiology;2022-12-20
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3