Cav1.2 and Cav1.3 L-type calcium channels independently control short- and long-term sensitization to pain-Reference-Cited by-同舟云学术

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Cav1.2 and Cav1.3 L-type calcium channels independently control short- and long-term sensitization to pain

Published:2016-07-03 Issue:22 Volume:594 Page:6607-6626
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Radwani Houda¹²³,Lopez-Gonzalez Maria José¹²,Cattaert Daniel¹⁴^ORCID,Roca-Lapirot Olivier¹²,Dobremez Eric¹⁵^ORCID,Bouali-Benazzouz Rabia¹²,Eiríksdóttir Emelía⁶,Langel Ülo⁶,Favereaux Alexandre¹²,Errami Mohammed³,Landry Marc¹²,Fossat Pascal¹²

Affiliation:

1. University of Bordeaux; Bordeaux France

2. CNRS, IINS, UMR5297; Bordeaux France

3. Faculty of Sciences; Abdelmalek Essaâdi University; Tetouan Morocco

4. CNRS, INCIA, UMR 5287; Talence France

5. Department of Pediatric Surgery; University Hospital of Bordeaux; Bordeaux France

6. Department of Neurochemistry and Neurotoxicology; Stockholm University; Stockholm Sweden

Funder

Conseil Général d'Aquitaine

Agence National pour la Recherche

Publisher

Wiley

Subject

Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1113/JP272725/fullpdf

Reference47 articles.

1. NMDA channels together with L-type calcium currents and calcium-activated nonspecific cationic currents are sufficient to generate windup in WDR neurons;Aguiar;J Neurophysiol,2010

2. Sensitization of pain pathways in the spinal cord: cellular mechanisms;Baranauskas;Prog Neurobiol,1998

3. Cellular and molecular mechanisms of pain;Basbaum;Cell,2009

4. Silencing of the Cav3.2 T-type calcium channel gene in sensory neurons demonstrates its major role in nociception;Bourinet;EMBO J,2005

5. Determinants of the G protein-dependent opioid modulation of neuronal calcium channels;Bourinet;Proc Natl Acad Sci USA,1996

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