Response to a Letter to the Editor: Physiological and pathophysiological roles of the KCNK3 potassium channel in the pulmonary circulation and the heart-Reference-Cited by-同舟云学术

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Response to a Letter to the Editor: Physiological and pathophysiological roles of the KCNK3 potassium channel in the pulmonary circulation and the heart

Published:2024-04-22 Issue:9 Volume:602 Page:2143-2144
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:The Journal of Physiology

Author:

Saint‐Martin Willer Anaïs¹²^ORCID,Capuano Véronique¹²,Montani David¹²³^ORCID,Antigny Fabrice¹²^ORCID

Affiliation:

1. Faculté de Médecine Université Paris‐Saclay Le Kremlin‐Bicêtre France

2. INSERM UMR_S 999 ‘Hypertension Pulmonaire: Physiopathologie et Innovation Thérapeutique’ Hôpital Marie Lannelongue Le Plessis‐Robinson France

3. Assistance Publique – Hôpitaux de Paris (AP‐HP), Service de Pneumologie et Soins Intensifs Respiratoires Centre de Référence de l'Hypertension Pulmonaire, Hôpital Bicêtre Le Kremlin‐Bicêtre France

Publisher

Wiley

Link

https://physoc.onlinelibrary.wiley.com/doi/pdf/10.1113/JP286650

Reference9 articles.

1. Potassium Channel Subfamily K Member 3 (KCNK3) Contributes to the Development of Pulmonary Arterial Hypertension

2. Loss of KCNK3 is a hallmark of RV hypertrophy/dysfunction associated with pulmonary hypertension

3. Characterization of Kcnk3 -Mutated Rat, a Novel Model of Pulmonary Hypertension

4. Impact of TASK-1 in Human Pulmonary Artery Smooth Muscle Cells

5. Comment on: ‘Physiological and pathophysiological roles of the KCNK3 potassium channel in the pulmonary circulation and the heart’ by Saint‐Martin Willer et al.

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