Novel mitochondrial transition pore inhibitor N ‐methyl‐4‐isoleucine cyclosporin is a new therapeutic option in acute pancreatitis-Reference-Cited by-同舟云学术

Novel mitochondrial transition pore inhibitor N ‐methyl‐4‐isoleucine cyclosporin is a new therapeutic option in acute pancreatitis

Published:2019-12 Issue:24 Volume:597 Page:5879-5898
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:The Journal of Physiology

Author:

Tóth Emese¹²,Maléth József¹³,Závogyán Noémi¹,Fanczal Júlia¹³^ORCID,Grassalkovich Anna¹²,Erdős Réka¹,Pallagi Petra¹³,Horváth Gergő⁴,Tretter László⁴,Bálint Emese Réka⁵^ORCID,Rakonczay Zoltán⁵,Venglovecz Viktória⁶,Hegyi Péter²⁷⁸^ORCID

Affiliation:

1. First Department of Medicine University of Szeged Szeged Hungary

2. Momentum Translational Gastroenterology Research Group Hungarian Academy of Sciences–University of Szeged Szeged Hungary

3. Momentum Epithelial Cell Signalling and Secretion Research Group Hungarian Academy of Sciences–University of Szeged Szeged Hungary

4. Department of Medical Biochemistry Semmelweis University Budapest Hungary

5. Department of Pathophysiology University of Szeged Szeged Hungary

6. Department of Pharmacology and Pharmacotherapy University of Szeged Szeged Hungary

7. Institute for Translational Medicine and First Department of Medicine University of Pécs Pécs Hungary

8. Szentágothai Research Centre University of Pécs Pécs Hungary

Funder

Magyar Tudományos Akadémia

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP278517

Reference62 articles.

1. Accelerating the Drug Delivery Pipeline for Acute and Chronic Pancreatitis

2. Resistance to cyclosporin A derives from mutations in hepatitis C virus nonstructural proteins

3. MORPHOLOGICAL, BIOCHEMICAL AND SECRETORY STUDIES ON RAT PANCREATIC DUCTS MAINTAINED IN TISSUE CULTURE

4. Effect of intravenous TRO40303 as an adjunct to primary percutaneous coronary intervention for acute ST-elevation myocardial infarction: MITOCARE study results

5. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

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