Control of vascular smooth muscle function by Src-family kinases and reactive oxygen species in health and disease
Author:
Affiliation:
1. Asthma, Allergy and Lung Biology, Faculty of Life Sciences and Medicine; King's College London; London UK
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2014.285304/fullpdf
Reference98 articles.
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2. Nitric oxide controls Src kinase activity through a sulfhydryl group modification-mediated Tyr-527-independent and Tyr-416-linked mechanism;Akhand;J Biol Chem,1999
3. Coupling of c-Src to large conductance voltage- and Ca2+-activated K+ channels as a new mechanism of agonist-induced vasoconstriction;Alioua;Proc Natl Acad Sci USA,2002
4. Src and Pyk2 mediate G-protein-coupled receptor activation of epidermal growth factor receptor (EGFR) but are not required for coupling to the mitogen-activated protein (MAP) kinase signaling cascade;Andreev;J Biol Chem,2001
5. Endothelin-1 activation of JAK2 in vascular smooth muscle cells involves NAD(P)H oxidase-derived reactive oxygen species;Banes-Berceli;Vascul Pharmacol,2005
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