Genetic ablation of ryanodine receptor 2 phosphorylation at Ser-2808 aggravates Ca2+-dependent cardiomyopathy by exacerbating diastolic Ca2+release-Reference-Cited by-同舟云学术

Genetic ablation of ryanodine receptor 2 phosphorylation at Ser-2808 aggravates Ca2+-dependent cardiomyopathy by exacerbating diastolic Ca2+release

Published:2014-02-21 Issue:9 Volume:592 Page:1957-1973
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Liu Bin¹,Ho Hsiang-Ting¹,Velez-Cortes Florencia¹,Lou Qing¹,Valdivia Carmen R.²,Knollmann Bjorn C.³,Valdivia Hector H.²,Gyorke Sandor¹

Affiliation:

1. Department of Physiology and Cell Biology; College of Medicine; and Davis Heart and Lung Research Institute; The Ohio State University; Columbus OH 43210 USA

2. Center for Arrhythmia Research; Cardiovascular Division of the Department of Internal Medicine; University of Michigan; Ann Arbor MI 48109 USA

3. Division of Clinical Pharmacology; Vanderbilt University Medical School; Nashville TN 37232 USA

Funder

National Institutes of Health

American Heart Association

Publisher

Wiley

Subject

Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2013.264689/fullpdf

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4. Fractional SR Ca release is regulated by trigger Ca and SR Ca content in cardiac myocytes;Bassani;Am J Physiol Cell Physiol,1995

5. The relationship between arrhythmogenesis and impaired contractility in heart failure: role of altered ryanodine receptor function;Belevych;Cardiovasc Res,2011

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