Huntingtin-associated protein 1 regulates exocytosis, vesicle docking, readily releasable pool size and fusion pore stability in mouse chromaffin cells-Reference-Cited by-同舟云学术

Huntingtin-associated protein 1 regulates exocytosis, vesicle docking, readily releasable pool size and fusion pore stability in mouse chromaffin cells

Published:2014-02-17 Issue:7 Volume:592 Page:1505-1518
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:The Journal of Physiology

Author:

Mackenzie Kimberly D.¹,Duffield Michael D.¹,Peiris Heshan¹,Phillips Lucy¹,Zanin Mark P.¹,Teo Ee Hiok¹,Zhou Xin-Fu¹²,Keating Damien J.¹

Affiliation:

1. Department of Human Physiology and Centre for Neuroscience; Flinders University; Adelaide SA Australia

2. Sansom Institute, University of South Australia; Adelaide SA Australia

Funder

Australian Research Council

National Health and Medical Research Council

Publisher

Wiley

Subject

Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2013.268342/fullpdf

Reference47 articles.

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2. F-actin and myosin II accelerate catecholamine release from chromaffin granules;Berberian;J Neurosci,2009

3. HAP1-huntingtin interactions do not contribute to the molecular pathology in Huntington's disease transgenic mice;Bertaux;FEBS Letters,1998

4. The R6/2 transgenic mouse model of Huntington's disease develops diabetes due to deficient β-cell mass and exocytosis;Bjorkqvist;Hum Mol Genet,2005

5. Fast transport and retrograde movement of huntingtin and HAP 1 in axons;Block-Galarza;Neuroreport,1997

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