Five ADNFLE Mutations Reduce the Ca2+Dependence of the Mammalian α4β2 Acetylcholine Response-Reference-Cited by-同舟云学术

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Five ADNFLE Mutations Reduce the Ca2+Dependence of the Mammalian α4β2 Acetylcholine Response

Published:2003-07 Issue:1 Volume:550 Page:11-26
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:The Journal of Physiology

Author:

Rodrigues‐Pinguet Nivalda¹²,Jia Li³,Li Maureen¹,Figl Antonio¹,Klaassen Alwin⁴,Truong Anthony¹,Lester Henry A.²,Cohen Bruce N.²

Affiliation:

1. Division of Biomedical Sciences, University of California, Riverside, CA 92521‐0121

2. Division of Biology, California Institute of Technology, Pasadena, CA 91125, USA

3. Computer Science Department, University of California, Riverside, CA 92521‐0121

4. Psychiatry and Biobehavioral Sciences, University of California, Los Angeles, CA 90095‐1759

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/jphysiol.2003.036681

Reference60 articles.

1. Mechanism for modulation of nicotinic acetylcholine receptors that can influence synaptic transmission;Amador M;J Neurosci,1995

2. Nucleotide sequence of the human nicotinic acetylcholine receptor β2 subunity gene

3. Relationship of sigma activity to sleep interictal epileptic discharges: a study in children affected by benign epilepsy with occipital paroxysms

4. How Mutations in the nAChRs Can Cause ADNFLE Epilepsy

5. Properties of neuronal nicotinic acetylcholine receptor mutants from humans suffering from autosomal dominant nocturnal frontal lobe epilepsy

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