An External Site Controls Closing of the Epithelial Na+Channel ENaC
Author:
Affiliation:
1. Institut de Pharmacologie et de Toxicologie, Bugnon 27, Université de Lausanne, CH‐1005 Lausanne, Switzerland
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1113/jphysiol.2002.022020
Reference35 articles.
1. Ripped Pocket and Pickpocket, Novel Drosophila DEG/ENaC Subunits Expressed in Early Development and in Mechanosensory Neurons
2. Protons Activate Brain Na+ Channel 1 by Inducing a Conformational Change That Exposes a Residue Associated with Neurodegeneration
3. Paradoxical Stimulation of a DEG/ENaC Channel by Amiloride
4. Mutations Causing Neurodegeneration in Caenorhabditis elegans Drastically Alter the pH Sensitivity and Inactivation of the Mammalian H+-gated Na+ Channel MDEG1
5. Protease Modulation of the Activity of the Epithelial Sodium Channel Expressed in Xenopus Oocytes
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