SOCs – Store‐Operated Channels in Vascular Smooth Muscle?
Author:
Affiliation:
1. School of Biomedical Sciences, University of Leeds, Leeds, UK
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1113/jphysiol.2002.027151
Reference6 articles.
1. A Ca 2+ ‐permeable non‐selective cation channel activated by depletion of internal Ca 2+ stores in single rabbit portal vein myocytes
2. Activation of store‐operated channels by noradrenaline via protein kinase C in rabbit portal vein myocytes
3. Exchange characteristics of the noradrenaline-sensitive calcium store in vascular smooth muscle cells or rabbit ear artery.
4. Activation of store-mediated calcium entry by secretion-like coupling between the inositol 1,4,5-trisphosphate receptor type II and human transient receptor potential (hTrp1) channels in human platelets
5. Calcium Influx Factor Directly Activates Store-operated Cation Channels in Vascular Smooth Muscle Cells
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2. Orai1 and Ca2+-independent phospholipase A2are required for store-operatedIcat-SOCcurrent, Ca2+entry, and proliferation of primary vascular smooth muscle cells;American Journal of Physiology-Cell Physiology;2012-03-01
3. Orai1, a critical component of store-operated Ca2+ entry, is functionally associated with Na+/Ca2+ exchanger and plasma membrane Ca2+ pump in proliferating human arterial myocytes;American Journal of Physiology-Cell Physiology;2009-11
4. Role of iPLA2 and store-operated channels in agonist-induced Ca2+ influx and constriction in cerebral, mesenteric, and carotid arteries;American Journal of Physiology-Heart and Circulatory Physiology;2008-03
5. α1-Adrenergic signaling mechanisms in contraction of resistance arteries;Reviews of Physiology, Biochemistry and Pharmacology
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