Selective mitochondrial Ca2+uptake deficit in disease endstage vulnerable motoneurons of the SOD1G93Amouse model of amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

Selective mitochondrial Ca2+uptake deficit in disease endstage vulnerable motoneurons of the SOD1G93Amouse model of amyotrophic lateral sclerosis

Published:2013-03-12 Issue:10 Volume:591 Page:2723-2745
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:

Author:

Fuchs Andrea,Kutterer Sylvie,Mühling Tobias,Duda Johanna,Schütz Burkhard,Liss Birgit,Keller Bernhard U.,Roeper Jochen

Publisher

Wiley

Subject

Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2012.247981/fullpdf

Reference72 articles.

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3. Development of hypoglossal motoneurons;Berger;J Appl Physiol,1996

4. Excitability properties of mouse motor axons in the mutant SOD1G93A model of amyotrophic lateral sclerosis;Boërio;Muscle Nerve,2010

5. Early electrophysiological abnormalities in lumbar motoneurons in a transgenic mouse model of amyotrophic lateral sclerosis;Bories;Eur J Neurosci,2007

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