Aldosterone responsiveness of the epithelial sodium channel (ENaC) in colon is increased in a mouse model for Liddle's syndrome
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2007.140459/fullpdf
Reference59 articles.
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3. Open probability of the epithelial sodium channel is regulated by intracellular sodium;Anantharam;J Physiol,2006
4. Aldosterone-induced increase in the abundance of Na+ channel subunits;Asher;Am J Physiol Cell Physiol,1996
5. Epithelial Na+ channel mutants causing Liddle's syndrome retain ability to respond to aldosterone and vasopressin;Auberson;Am J Physiol Renal Physiol,2003
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