Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE2production and increased heat loss-Reference-Cited by-同舟云学术

Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE2production and increased heat loss

Published:2020-08-21 Issue:20 Volume:598 Page:4663-4680
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Amorim Mateus R.¹²^ORCID,Moreira Diego A.¹^ORCID,Santos Bruna M.³,Ferrari Gustavo D.³⁴,Nogueira Jonatas E.⁵,de Deus Júnia L.¹⁶,Alberici Luciane C.⁴^ORCID,Branco Luiz G. S.¹^ORCID

Affiliation:

1. Dental School of Ribeirão Preto University of São Paulo Ribeirão Preto São Paulo Brazil

2. Division of Pulmonary and Critical Care Medicine, Department of Medicine Johns Hopkins University, School of Medicine Baltimore MD USA

3. Medical School of Ribeirão Preto University of São Paulo Ribeirão Preto São Paulo Brazil

4. Department of Biomolecular Sciences, School of Pharmaceutical Sciences of Ribeirão Preto University of São Paulo Ribeirão Preto São Paulo Brazil

5. School of Physical Education and Sports of Ribeirão Preto University of São Paulo Ribeirão Preto SP Brazil

6. The Solomon H. Snyder. Department of Neuroscience Johns Hopkins University, School of Medicine Baltimore MD USA

Funder

Fundação de Amparo à Pesquisa do Estado de São Paulo

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP280321

Reference69 articles.

1. Hypertension is critically dependent on the carotid body input in the spontaneously hypertensive rat

2. Lipopolysaccharide-induced hypothermia and hypotension are associated with inflammatory signaling that is triggered outside the brain

3. Oxidative Stress and Antioxidant Activity in Hypothermia and Rewarming: Can RONS Modulate the Beneficial Effects of Therapeutic Hypothermia?

4. Is Cardiac Hypertrophy in Spontaneously Hypertensive Rats the Cause or the Consequence of Oxidative Stress?

5. Role of respiratory changes in the modulation of arterial pressure in rats submitted to sino-aortic denervation

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