CaMKII does not control mitochondrial Ca 2+ uptake in cardiac myocytes-Reference-Cited by-同舟云学术

CaMKII does not control mitochondrial Ca 2+ uptake in cardiac myocytes

Published:2020-04 Issue:7 Volume:598 Page:1361-1376
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Nickel Alexander G.¹²,Kohlhaas Michael¹²,Bertero Edoardo¹,Wilhelm Daniel²^ORCID,Wagner Michael²³,Sequeira Vasco¹,Kreusser Michael M.⁴⁵⁶,Dewenter Matthias⁴⁵,Kappl Reinhard⁷,Hoth Markus⁷^ORCID,Dudek Jan¹,Backs Johannes⁴⁵,Maack Christoph¹^ORCID

Affiliation:

1. Comprehensive Heart Failure Center (CHFC)University Clinic Würzburg Würzburg Germany

2. Affiliation when/at which experiments were performed: Clinic III for Internal MedicineUniversity Clinic Homburg Homburg Germany

3. Institute for Molecular Cell BiologySaarland University Homburg Germany

4. Institute of Experimental CardiologyHeidelberg University Hospital Heidelberg Germany

5. German Center for Cardiovascular Research (DZHK)partner site Heidelberg/Mannheim Germany

6. Department of CardiologyHeidelberg University Hospital Heidelberg Germany

7. Department of BiophysicsCIPMM, School of Medicine, Saarland University Homburg Germany

Funder

European Commission

Bundesministerium für Bildung und Forschung

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP276766

Reference48 articles.

1. CaMKII in myocardial hypertrophy and heart failure

2. Synchronized Whole Cell Oscillations in Mitochondrial Metabolism Triggered by a Local Release of Reactive Oxygen Species in Cardiac Myocytes

3. The isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload

4. CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy

5. Cardiac Energy Metabolism Homeostasis: Role of Cytosolic Calcium

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