Contraction‐regulated mTORC1 and protein synthesis: Influence of AMPK and glycogen
Author:
Affiliation:
1. Section of Molecular PhysiologyDepartment of NutritionExercise and SportsUniversity of Copenhagen Copenhagen Denmark
Funder
Novo Nordisk Fonden
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP279780
Reference58 articles.
1. Resistance exercise-induced S6K1 kinase activity is not inhibited in human skeletal muscle despite prior activation of AMPK by high-intensity interval cycling
2. Resistance exercise induced mTORC1 signaling is not impaired by subsequent endurance exercise in human skeletal muscle
3. mTORC1 signaling and the metabolic control of cell growth
4. Differential response of skeletal muscles to mTORC1 signaling during atrophy and hypertrophy
5. Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo
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