Intercellular model predicts mechanisms of inflammation‐fibrosis coupling after myocardial infarction-Reference-Cited by-同舟云学术

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Intercellular model predicts mechanisms of inflammation‐fibrosis coupling after myocardial infarction

Published:2022-08-08 Issue: Volume: Page:
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:The Journal of Physiology

Author:

Chowkwale Mukti¹^ORCID,Lindsey Merry L.²³,Saucerman Jeffrey J.¹⁴

Affiliation:

1. Department of Biomedical Engineering University of Virginia Charlottesville VA USA

2. School of Graduate Studies and Research Meharry Medical College Nashville TN USA

3. Research Service Nashville VA Medical Center Nashville TN USA

4. Robert M. Berne Cardiovascular Research Center University of Virginia Charlottesville VA USA

Funder

National Institutes of Health

U.S. Department of Veterans Affairs

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP283346

Reference82 articles.

1. Anakinra, a Recombinant Human Interleukin-1 Receptor Antagonist, Inhibits Apoptosis in Experimental Acute Myocardial Infarction

2. Principles of Cell Circuits for Tissue Repair and Fibrosis

3. Azithromycin therapy reduces cardiac inflammation and mitigates adverse cardiac remodeling after myocardial infarction: Potential therapeutic targets in ischemic heart disease

4. The infarcted myocardium solicits GM-CSF for the detrimental oversupply of inflammatory leukocytes

5. Nanoparticle Delivery of miRNA-21 Mimic to Cardiac Macrophages Improves Myocardial Remodeling after Myocardial Infarction

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