Long QT syndrome caveolin‐3 mutations differentially modulate K v 4 and Ca v 1.2 channels to contribute to action potential prolongation-Reference-Cited by-同舟云学术

Long QT syndrome caveolin‐3 mutations differentially modulate K v 4 and Ca v 1.2 channels to contribute to action potential prolongation

Published:2019-01-24 Issue:6 Volume:597 Page:1531-1551
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Tyan Leonid¹,Foell Jason D.¹,Vincent Kevin P.²,Woon Marites T.¹,Mesquitta Walatta T.¹,Lang Di¹,Best Jabe M.¹,Ackerman Michael J.³,McCulloch Andrew D.²⁴^ORCID,Glukhov Alexey V.¹^ORCID,Balijepalli Ravi C.¹,Kamp Timothy J.¹^ORCID

Affiliation:

1. Department of MedicineDivision of Cardiovascular MedicineUniversity of Wisconsin‐Madison 1111 Highland Ave Madison WI USA

2. Department of BioengineeringUniversity of California San Diego 9500 Gilman Drive La Jolla CA USA

3. Departments of Cardiovascular MedicinePediatric and Adolescent Medicine and Molecular Pharmacology & Experimental TherapeuticsDivisions of Heart Rhythm Services and Pediatric CardiologyWindland Smith Rice Sudden Death Genomics LaboratoryMayo Clinic 200 First Street SW Rochester MN USA

4. Department of MedicineUniversity of California San Diego 9500 Gilman Drive La Jolla CA USA

Funder

National Institutes of Health

American Heart Association

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP276014

Reference46 articles.

1. α1-Adrenoreceptors regulate only the caveolae-located subpopulation of cardiac KV4 channels