Amyloid Beta42 oligomers up‐regulate the excitatory synapses by potentiating presynaptic release while impairing postsynaptic NMDA receptors-Reference-Cited by-同舟云学术

Amyloid Beta42 oligomers up‐regulate the excitatory synapses by potentiating presynaptic release while impairing postsynaptic NMDA receptors

Published:2020-05-14 Issue:11 Volume:598 Page:2183-2197
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Marcantoni Andrea¹^ORCID,Cerullo Maria Sabina¹^ORCID,Buxeda Pol¹,Tomagra Giulia¹^ORCID,Giustetto Maurizio²³^ORCID,Chiantia Giuseppe¹^ORCID,Carabelli Valentina¹^ORCID,Carbone Emilio¹^ORCID

Affiliation:

1. Department of Drug Science and Technology Torino University Italy

2. Department of Neurosciences / National Institute of Neuroscience Torino University Italy

3. National Institute of Neuroscience‐Italy Turin Italy

Funder

Ministero dell’Istruzione, dell’Università e della Ricerca

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP279345

Reference51 articles.

1. Bud extracts from Tilia tomentosa Moench inhibit hippocampal neuronal firing through GABAA and benzodiazepine receptors activation

2. Synaptic and intrinsic mechanisms shape synchronous oscillations in hippocampal neurons in culture

3. Brain-Derived Neurotrophic Factor Enhances GABA Release Probability and Nonuniform Distribution of N- and P/Q-Type Channels on Release Sites of Hippocampal Inhibitory Synapses

4. BDNF up-regulates evoked GABAergic transmission in developing hippocampus by potentiating presynaptic N- and P/Q-type Ca2+channels signalling

5. NMDA and non-NMDA receptors are co-localized at individual excitatory synapses in cultured rat hippocampus

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