Increased sarcoplasmic/endoplasmic reticulum calcium ATPase 2a activity underlies the mechanism of the positive inotropic effect of ivabradine-Reference-Cited by-同舟云学术

Increased sarcoplasmic/endoplasmic reticulum calcium ATPase 2a activity underlies the mechanism of the positive inotropic effect of ivabradine

Published:2020-01-29 Issue:3 Volume:105 Page:477-488
ISSN:0958-0670
Container-title:Experimental Physiology
language:en
Short-container-title:Exp Physiol

Author:

Xie Ming¹²^ORCID,Huang Hui‐Li²,Zhang Wen‐Hui²,Gao Li²,Wang Yu‐Wei²,Zhu Xiao‐Jia²,Li Wei²,Chen Ke‐Su³,Boutjdir Mohamed⁴⁵⁶,Chen Long²⁷^ORCID

Affiliation:

1. Department of PharmacyJiangyin Hospital of TCM Affiliated to Nanjing University of Chinese Medicine Jiangyin China

2. Jiangsu key Laboratory for Pharmacology and Safety Evaluation of Chinese Materia MedicaSchool of PharmacyNanjing University of Chinese Medicine Nanjing China

3. Department of RespiratoryInpatient Wards for Senior CadresGeneral Hospital of Eastern Theater CommandPLA Nanjing China

4. VA New York Harbor Healthcare System 800 Poly place Brooklyn NY USA

5. State University of New York Downstate Medical Center 450 Clarkson Avenue Brooklyn NY USA

6. NYU School of Medicine 550 First Avenue New York NY USA

7. Institute of Chinese Medicine of Taizhou China Medical City Double Tower, China Medical City Taizhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Physiology,Physiology (medical),Nutrition and Dietetics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/EP087964

Reference34 articles.

1. Phosphodiesterase 4D Regulates Baseline Sarcoplasmic Reticulum Ca 2+ Release and Cardiac Contractility, Independently of L-Type Ca 2+ Current

2. Calcium Cycling and Signaling in Cardiac Myocytes

3. Noninvasive evaluation of left ventricular elastance according to pressure-volume curves modeling in arterial hypertension

4. Voltage-clamp investigations of membrane currents underlying pace-maker activity in rabbit sino-atrial node.

5. α 1 -Adrenergic Activation Inhibits β-Adrenergic–Stimulated Unitary Ca 2+ Currents in Cardiac Ventricular Myocytes

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