Paving the way for Bartter syndrome type 3 drug discovery: a hope from basic research
Author:
Affiliation:
1. Department of Pharmacy-Drug Sciences; University of Bari; Bari Italy
Funder
Fondazione Cassa di Risparmio di Puglia
Fondazione Telethon
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/JP274645/fullpdf
Reference6 articles.
1. Functional severity of CLCNKB mutations correlates with phenotypes in patients with classic Bartter's syndrome;Cheng;J Physiol,2017
2. Barttin is a Cl− channel β-subunit crucial for renal Cl− reabsorption and inner ear K+ secretion;Estévez;Nature,2001
3. Targeting kidney CLC-K channels: pharmacological profile in a human cell line versus Xenopus oocytes;Imbrici;Biochim Biophys Acta,2014
4. Pharmacovigilance database search discloses ClC-K channels as a novel target of the AT1 receptor blockers valsartan and olmesartan;Imbrici;Br J Pharmacol,2017
5. Structure of a CLC chloride ion channel by cryo-electron microscopy;Park;Nature,2017
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Bartter Syndrome-Related Variants Distribution: Brazilian Data and Its Comparison with Worldwide Cohorts;Nephron;2023
2. Functional Study of Novel Bartter’s Syndrome Mutations in ClC-Kb and Rescue by the Accessory Subunit Barttin Toward Personalized Medicine;Frontiers in Pharmacology;2020-03-17
3. Ion Channels in Drug Discovery and Safety Pharmacology;Methods in Molecular Biology;2018
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