Adaptive exhaustion during prolonged intermittent hypoxia causes dysregulated skeletal muscle protein homeostasis-Reference-Cited by-同舟云学术

Adaptive exhaustion during prolonged intermittent hypoxia causes dysregulated skeletal muscle protein homeostasis

Published:2023-01-10 Issue:3 Volume:601 Page:567-606
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:The Journal of Physiology

Author:

Attaway Amy H.¹,Bellar Annette²,Mishra Saurabh²,Karthikeyan Manikandan²,Sekar Jinendiran²,Welch Nicole²³,Musich Ryan²,Singh Shashi Shekhar²^ORCID,Kumar Avinash²,Menon Aishwarya²,King Jasmine²,Langen Ramon⁴,Webster Justine⁴,Scheraga Rachel G.¹²,Rochon Kristy⁵^ORCID,Mears Jason⁵^ORCID,Naga Prasad Sathyamangla V.⁶,Hatzoglou Maria⁷,Chakraborty Abhishek A.⁸,Dasarathy Srinivasan²³^ORCID

Affiliation:

1. Department of Pulmonary Medicine Respiratory Institute Cleveland Clinic Cleveland Ohio USA

2. Department of Inflammation and Immunity Lerner Research Institute Cleveland Clinic Cleveland Ohio USA

3. Department of Gastroenterology and Hepatology Cleveland Clinic Cleveland Ohio USA

4. Department of Respiratory Medicine Maastricht University Medical Center Maastricht Netherlands

5. Department of Pharmacology Case Western Reserve University Cleveland Ohio USA

6. Department of Cardiovascular and Metabolic Diseases Lerner Research Institute Cleveland Clinic Cleveland Ohio USA

7. Department of Genomic Medicine Case Western Reserve University Cleveland Ohio USA

8. Department of Cancer Biology Lerner Research Institute Cleveland Clinic Cleveland Ohio USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP283700

Reference101 articles.

1. Three Distinct Mechanisms Generate Oxygen Free Radicals in Neurons and Contribute to Cell Death during Anoxia and Reoxygenation

2. Blocked O-GlcNAc cycling alters mitochondrial morphology, function, and mass

3. Tutorial on Protein Ontology Resources

4. Quantifying Mitochondrial Dynamics in Patient Fibroblasts with Multiple Developmental Defects and Mitochondrial Disorders

5. Sex Differences in Chronic Obstructive Pulmonary Disease Mechanisms

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