K2PTASK-2 and KCNQ1-KCNE3 K+channels are major players contributing to intestinal anion and fluid secretion-Reference-Cited by-同舟云学术

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K2PTASK-2 and KCNQ1-KCNE3 K+channels are major players contributing to intestinal anion and fluid secretion

Published:2017-12-18 Issue:3 Volume:596 Page:393-407
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Julio-Kalajzić Francisca¹,Villanueva Sandra¹²,Burgos Johanna¹²,Ojeda Margarita¹,Cid L. Pablo¹,Jentsch Thomas J.³,Sepúlveda Francisco V.¹^ORCID

Affiliation:

1. Centro de Estudios Científicos (CECs); Avenida Arturo Prat 514 Valdivia Chile

2. Universidad Austral de Chile; Valdivia Chile

3. Leibniz-Institut für Molekulare Pharmakologie (FMP) and Max-Delbrück-Centrum für Molekulare Medizin (MDC); Berlin Germany

Funder

Conicyt-Chile Basal Financing Programme

Fondecyt-Chile

Fondo Nacional de Desarrollo Científico y Tecnológico

Publisher

Wiley

Subject

Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1113/JP275178/fullpdf

Reference56 articles.

1. Disease-associated mutations in KCNE potassium channel subunits (MiRPs) reveal promiscuous disruption of multiple currents and conservation of mechanism;Abbott;FASEB J,2002

2. cAMP and Ca2+ signaling in secretory epithelia: crosstalk and synergism;Ahuja;Cell Calcium,2014

3. Upregulation of basolateral small conductance potassium channels (KCNQ1/KCNE3) in ulcerative colitis;Al-Hazza;Biochem Biophys Res Commun,2016

4. Potential role of reduced basolateral potassium (IKCa3.1) channel expression in the pathogenesis of diarrhoea in ulcerative colitis;Al-Hazza;J Pathol,2012

5. Berberine reduces cAMP-induced chloride secretion in T84 human colonic carcinoma cells through inhibition of basolateral KCNQ1 channels;Alzamora;Front Physiol,2011

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