Use of genetic models of idiopathic hypogonadotrophic hypogonadism in mice and men to understand the mechanisms of disease
Author:
Publisher
Wiley
Subject
Physiology,Physiology (medical),Nutrition and Dietetics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/expphysiol.2013.071910/fullpdf
Reference44 articles.
1. Human GnRH deficiency: a unique disease model to unravel the ontogeny of GnRH neurons;Balasubramanian;Neuroendocrinology,2010
2. A novel severe N-terminal splice site KISS1R gene mutation causes hypogonadotropic hypogonadism but enables a normal development of neonatal external genitalia;Breuer;Eur J Endocrinol,2012
3. Two families with normosmic congenital hypogonadotropic hypogonadism and biallelic mutations in KISS1R (KISS1 receptor): clinical evaluation and molecular characterization of a novel mutation;Brioude;PLoS One,2013
4. GnRH-deficient phenotypes in humans and mice with heterozygous variants in KISS1/Kiss1;Chan;J Clin Endocrinol Metab,2011
5. Kisspeptin/Gpr54-independent gonadotrophin-releasing hormone activity in Kiss1 and Gpr54 mutant mice;Chan;J Neuroendocrinol,2009
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