Cell volume control in phospholemman (PLM) knockout mice: do cardiac myocytes demonstrate a regulatory volume decrease and is this influenced by deletion of PLM?
Author:
Publisher
Wiley
Subject
Physiology,Physiology (medical),Nutrition and Dietetics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/expphysiol.2008.045823/fullpdf
Reference44 articles.
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2. KVLQT1 and IsK (minK) proteins associate to form the IKS cardiac potassium current;Barhanin;Nature,1996
3. Phospholemman transmembrane structure reveals potential interactions with Na+/K+-ATPase;Beevers;J Biol Chem,2007
4. Characterization of the phospholemman knockout mouse heart: depressed left ventricular function with increased Na-K-ATPase activity;Bell;Am J Physiol Heart Circ Physiol,2008
5. The minK potassium channel exists in functional and nonfunctional forms when expressed in the plasma membrane of Xenopus oocytes;Blumenthal;J Neurosci,1994
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4. Hypotonic swelling promotes nitric oxide release in cardiac ventricular myocytes: impact on swelling-induced negative inotropic effect;Cardiovascular Research;2014-10-24
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