Interleukin-1β blockade improves cardiac remodelling after myocardial infarction without interrupting the inflammasome in the mouse-Reference-Cited by-同舟云学术

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Interleukin-1β blockade improves cardiac remodelling after myocardial infarction without interrupting the inflammasome in the mouse

Published:2013-01-21 Issue:3 Volume:98 Page:734-745
ISSN:0958-0670
Container-title:Experimental Physiology
language:en
Short-container-title:

Author:

Toldo Stefano,Mezzaroma Eleonora,Van Tassell Benjamin W.,Farkas Daniela,Marchetti Carlo,Voelkel Norbert F.,Abbate Antonio

Publisher

Wiley

Subject

Physiology,Physiology (medical),Nutrition and Dietetics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1113/expphysiol.2012.069831/fullpdf

Reference38 articles.

1. The heart on fire: inflammasome and cardiomyopathy;Abbate;Exp Physiol,2012

2. Pathophysiologic role of myocardial apoptosis in post-infarction left ventricular remodeling;Abbate;J Cell Physiol,2002a

3. Increased myocardial apoptosis in patients with unfavorable left ventricular remodeling and early symptomatic post-infarction heart failure;Abbate;J Am Coll Cardiol,2003

4. Persistent infarct-related artery occlusion is associated with an increased myocardial apoptosis at postmortem examination in humans late after an acute myocardial infarction;Abbate;Circulation,2002b

5. Infarct-related artery occlusion, tissue markers of ischaemia, and increased apoptosis in the peri-infarct viable myocardium;Abbate;Eur Heart J,2005

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