Reply from David P. Burns, Eric F. Lucking and Ken D. O'Halloran: Auxiliary compensation for diaphragm dysfunction in dystrophic disease
Author:
Affiliation:
1. Department of PhysiologySchool of MedicineCollege of Medicine and HealthUniversity College Cork Cork Ireland
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP278371
Reference17 articles.
1. Length and curvature of the dog diaphragm
2. Kinematics and mechanics of midcostal diaphragm of dog
3. Recovery of respiratory function in mdx mice co-treated with neutralizing interleukin-6 receptor antibodies and urocortin-2
4. Inspiratory pressure‐generating capacity is preserved during ventilatory and non‐ventilatory behaviours in young dystrophic mdx mice despite profound diaphragm muscle weakness
5. Sensorimotor control of breathing in themdxmouse model of Duchenne muscular dystrophy
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Loss of compensation afforded by accessory muscles of breathing leads to respiratory system compromise in the mdx mouse model of Duchenne muscular dystrophy;The Journal of Physiology;2023-09-08
2. Loss of compensation by accessory muscles of breathing in advanced dystrophic disease;The FASEB Journal;2021-05
3. Contribution of extra‐diaphragmatic inspiratory muscles to peak inspiratory pressure in wild‐type and dystrophic ( mdx ) mice;The FASEB Journal;2021-05
4. N-acetylcysteine Decreases Fibrosis and Increases Force-Generating Capacity of mdx Diaphragm;Antioxidants;2019-11-24
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