Cerebellar ataxias: β-III spectrin's interactions suggest common pathogenic pathways
Author:
Affiliation:
1. Centre for Integrative Physiology; University of Edinburgh; Hugh Robson Building, George Square Edinburgh EH8 9XD UK
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/JP271195/fullpdf
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3. Mutant β-III spectrin causes mGluR1α mislocalization and functional deficits in a mouse model of spinocerebellar ataxia type 5;Armbrust;J Neurosci,2014
4. Prodynorphin mutations cause the neurodegenerative disorder spinocerebellar ataxia type 23;Bakalkin;Am J Hum Genet,2010
5. Intracellular FGF14 (iFGF14) is required for spontaneous and evoked firing in cerebellar Purkinje neurons and for motor coordination and balance;Bosch;J Neurosci,2015
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