The roles of c-Jun NH2-terminal kinases (JNKs) in obesity and insulin resistance
Author:
Affiliation:
1. Division of Diabetes and Metabolism; Garvan Institute of Medical Research; Darlinghurst Sydney Australia
2. Cellular & Molecular Metabolism Laboratory; Baker IDI Heart and Diabetes Institute; Melbourne Australia
Publisher
Wiley
Subject
Physiology
Reference65 articles.
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2. The c-Jun amino-terminal kinase pathway is preferentially activated by interleukin-1 and controls apoptosis in differentiating pancreatic β-cells;Ammendrup;Diabetes,2000
3. Hypothalamic and pituitary c-Jun N-terminal kinase 1 signaling coordinately regulates glucose metabolism;Belgardt;Proc Natl Acad Sci USA,2010
4. Increase in endoplasmic reticulum stress-related proteins and genes in adipose tissue of obese, insulin-resistant individuals;Boden;Diabetes,2008
5. IKKβ/NF-κB activation causes severe muscle wasting in mice;Cai;Cell,2004
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