Mechanisms underlying selective coupling of endothelial Ca 2+ signals with eNOS vs . IK/SK channels in systemic and pulmonary arteries-Reference-Cited by-同舟云学术

Mechanisms underlying selective coupling of endothelial Ca 2+ signals with eNOS vs . IK/SK channels in systemic and pulmonary arteries

Published:2020-06-11 Issue:17 Volume:598 Page:3577-3596
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Ottolini Matteo¹²,Daneva Zdravka¹,Chen Yen‐Lin¹,Cope Eric L.¹,Kasetti Ramesh B.³,Zode Gulab S.³,Sonkusare Swapnil K.¹²⁴^ORCID

Affiliation:

1. Robert M. Berne Cardiovascular Research Center University of Virginia‐School of Medicine Charlottesville VA USA

2. Department of Pharmacology University of Virginia‐School of Medicine Charlottesville VA USA

3. Department of Pharmacology and Neuroscience and the North Texas Eye Research Institute University of North Texas Health Science Center Fort Worth TX USA

4. Department of Molecular Physiology and Biological Physics University of Virginia‐School of Medicine Charlottesville VA USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP279570

Reference68 articles.

1. Targeting Pulmonary Endothelial Hemoglobin α Improves Nitric Oxide Signaling and Reverses Pulmonary Artery Endothelial Dysfunction

2. RSK2 contributes to myogenic vasoconstriction of resistance arteries by activating smooth muscle myosin and the Na + /H + exchanger

3. Low intravascular pressure activates endothelial cell TRPV4 channels, local Ca2+ events, and IKCa channels, reducing arteriolar tone

4. On the local reactions of the arterial wall to changes of internal pressure

5. Dissecting the molecular control of endothelial NO synthase by caveolin-1 using cell-permeable peptides

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