Implication of ZNF217 in Accelerating Tumor Development and Therapeutically Targeting ZNF217-Induced PI3K–AKT Signaling for the Treatment of Metastatic Osteosarcoma
Author:
Funder
NIH
NIHNIAMSNIAMS
NIHNIAIDNIAID
Publisher
American Association for Cancer Research (AACR)
Subject
Cancer Research,Oncology
Cited by 12 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Hsa_circ_0049472 contributed to amyloid-beta peptide-induced neurotoxicity, apoptosis and inflammation via regulating PI3K-AKT signaling pathway by interacting with miR-22–3p/ZNF217 axis;Brain Research Bulletin;2024-09
2. Suppression of N-Glycosylation of Zinc Finger Protein 471 Affects Proliferation, Invasion, and Docetaxel Sensitivity of Tongue Squamous Cell Carcinoma via Regulation of c-Myc;The American Journal of Pathology;2024-06
3. ZNF217: An Oncogenic Transcription Factor and Potential Therapeutic Target for Multiple Human Cancers;Cancer Management and Research;2024-01
4. Genomic and Molecular Characteristics of Ovarian Carcinosarcoma;American Journal of Clinical Oncology;2023-10-17
5. Dimethyl fumarate inhibits ZNF217 and can be beneficial in a subset of estrogen receptor positive breast cancers;Breast Cancer Research and Treatment;2023-07-21
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