Dual Targeting of Aurora Kinases with AMG 900 Exhibits Potent Preclinical Activity Against Acute Myeloid Leukemia with Distinct Post-Mitotic Outcomes-Reference-Cited by-同舟云学术

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Dual Targeting of Aurora Kinases with AMG 900 Exhibits Potent Preclinical Activity Against Acute Myeloid Leukemia with Distinct Post-Mitotic Outcomes

Published:2018-09-28 Issue:12 Volume:17 Page:2575-2585
ISSN:1535-7163
Container-title:Molecular Cancer Therapeutics
language:en
Short-container-title:Mol Cancer Ther

Author:

Payton Marc,Cheung Hung-Kam,Ninniri Maria Stefania S.,Marinaccio Christian,Wayne William C.,Hanestad Kelly,Crispino John D.,Juan Gloria,Coxon Angela

Funder

NIH

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 13 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Calreticulin and JAK2V617F driver mutations induce distinct mitotic defects in myeloproliferative neoplasms;Scientific Reports;2024-02-02

2. Rationale, in silico docking, ADMET profile, design, synthesis and cytotoxicity evaluations of phthalazine derivatives as VEGFR-2 inhibitors and apoptosis inducers;RSC Advances;2024

3. Small-molecule inhibition of kinesin KIF18A reveals a mitotic vulnerability enriched in chromosomally unstable cancers;Nature Cancer;2023-12-27

4. Calreticulin and JAK2V617F driver mutations induce distinct mitotic defects in myeloproliferative neoplasms;2023-10-31

5. Concomitant targeting of FLT3 and BTK overcomes FLT3 inhibitor resistance in acute myeloid leukemia through inhibition of autophagy;Haematologica;2022-10-13

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