TRP53 Mutants Drive Neuroendocrine Lung Cancer Through Loss-of-Function Mechanisms with Gain-of-Function Effects on Chemotherapy Response-Reference-Cited by-同舟云学术

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TRP53 Mutants Drive Neuroendocrine Lung Cancer Through Loss-of-Function Mechanisms with Gain-of-Function Effects on Chemotherapy Response

Published:2017-08-28 Issue:12 Volume:16 Page:2913-2926
ISSN:1535-7163
Container-title:Molecular Cancer Therapeutics
language:en
Short-container-title:Mol Cancer Ther

Author:

Akeno Nagako,Reece Alisa L.,Callahan Melissa,Miller Ashley L.,Kim Rebecca G.,He Diana,Lane Adam,Moulton Jonathan S.,Wikenheiser-Brokamp Kathryn A.

Funder

National Heart Lung and Blood Institute

American Cancer Society

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 13 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Targeting Oncogenic Mutant p53 and BCL-2 for Small Cell Lung Cancer Treatment;International Journal of Molecular Sciences;2023-08-23

2. Further evidence of an evolutionary continuum from a subset of lung carcinoids to aggressive neuroendocrine tumors;2023-03-20

3. Binary pan-cancer classes with distinct vulnerabilities defined by pro- or anti-cancer YAP/TEAD activity;Cancer Cell;2021-08

4. Characterizing TP53 mutations in ovarian carcinomas with and without concurrent BRCA1 or BRCA2 mutations;Gynecologic Oncology;2021-03

5. Mutant p53 promotes RCP-dependent chemoresistance coinciding with increased delivery of P-glycoprotein to the plasma membrane;Cell Death & Disease;2021-02

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