FGFR-Mediated Reactivation of MAPK Signaling Attenuates Antitumor Effects of Imatinib in Gastrointestinal Stromal Tumors-Reference-Cited by-同舟云学术

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FGFR-Mediated Reactivation of MAPK Signaling Attenuates Antitumor Effects of Imatinib in Gastrointestinal Stromal Tumors

Published:2015-02-11 Issue:4 Volume:5 Page:438-451
ISSN:2159-8274
Container-title:Cancer Discovery
language:en
Short-container-title:Cancer Discovery

Author:

Li Fang,Huynh Hung,Li Xiaoyan,Ruddy David A.,Wang Youzhen,Ong Richard,Chow Pierce,Qiu Shumei,Tam Angela,Rakiec Daniel P.,Schlegel Robert,Monahan John E.,Huang Alan

Publisher

American Association for Cancer Research (AACR)

Subject

Oncology

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1. Fibroblast Growth Factor 2 (FGF2) Activates Vascular Endothelial Growth Factor (VEGF) Signaling in Gastrointestinal Stromal Tumors (GIST): An Autocrine Mechanism Contributing to Imatinib Mesylate (IM) Resistance;Cancers;2024-09-07

2. Triple combination therapy comprising osimertinib, an AXL inhibitor, and an FGFR inhibitor improves the efficacy of EGFR-mutated non-small cell lung cancer;Cancer Letters;2024-08

3. From Tumor to Bone: Growth Factor Receptors as Key Players in Cancer Metastasis;Frontiers in Bioscience-Landmark;2024-05-13

4. Clinical Efficacy and Mechanism of Action of Recently FDA Approved Anticancer Drugs: An Updated Review;Current Cancer Therapy Reviews;2024-04-09

5. KIT mutations and expression: current knowledge and new insights for overcoming IM resistance in GIST;Cell Communication and Signaling;2024-02-27

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