SGK1 Is a Critical Component of an AKT-Independent Pathway Essential for PI3K-Mediated Tumor Development and Maintenance-Reference-Cited by-同舟云学术

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SGK1 Is a Critical Component of an AKT-Independent Pathway Essential for PI3K-Mediated Tumor Development and Maintenance

Published:2017-10-20 Issue:24 Volume:77 Page:6914-6926
ISSN:0008-5472
Container-title:Cancer Research
language:en
Short-container-title:Cancer Res

Author:

Orlacchio Arturo,Ranieri Michela,Brave Martina,Arciuch Valeria Antico,Forde Toni,De Martino Daniela,Anderson Karen E.,Hawkins Phillip,Di Cristofano Antonio

Funder

Albert Einstein Cancer Center

NIH

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 31 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. CD133-Dependent Activation of Phosphoinositide 3-Kinase /AKT/Mammalian Target of Rapamycin Signaling in Melanoma Progression and Drug Resistance;Cells;2024-01-26

2. Matrix stiffness influences response to chemo and targeted therapy in brain metastatic breast cancer cells;Biomaterials Science;2024

3. The choreography of protein kinase PDK1 and its diverse substrate dance partners;Biochemical Journal;2023-10-04

4. AKT-independent signaling in PIK3CA-mutant thyroid cancer mediates resistance to dual SRC and MEK1/2 inhibition;Medical Oncology;2023-09-15

5. Luteolin inhibits triple-negative breast cancer by inducing apoptosis and autophagy through SGK1-FOXO3a-BNIP3 signaling;Frontiers in Pharmacology;2023-06-06

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