Enhanced Histone Deacetylase Activity in Malignant Melanoma Provokes RAD51 and FANCD2-Triggered Drug Resistance-Reference-Cited by-同舟云学术

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Enhanced Histone Deacetylase Activity in Malignant Melanoma Provokes RAD51 and FANCD2-Triggered Drug Resistance

Published:2016-03-15 Issue:10 Volume:76 Page:3067-3077
ISSN:0008-5472
Container-title:Cancer Research
language:en
Short-container-title:Cancer Res

Author:

Krumm Andrea,Barckhausen Christina,Kücük Pelin,Tomaszowski Karl-Heinz,Loquai Carmen,Fahrer Jörg,Krämer Oliver Holger,Kaina Bernd,Roos Wynand Paul

Funder

German Research Foundation

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 72 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. GSE1 links the HDAC1/CoREST co-repressor complex to DNA damage;Nucleic Acids Research;2023-10-25

2. Exploring the role of epigenetic alterations and non-coding RNAs in melanoma pathogenesis and therapeutic strategies;Melanoma Research;2023-10-02

3. RNA methylation, homologous recombination repair and therapeutic resistance;Biomedicine & Pharmacotherapy;2023-10

4. Class I HDAC Inhibition Leads to a Downregulation of FANCD2 and RAD51, and the Eradication of Glioblastoma Cells;Journal of Personalized Medicine;2023-08-27

5. Histone Deacetylases (HDAC) Inhibitor—Valproic Acid Sensitizes Human Melanoma Cells to Dacarbazine and PARP Inhibitor;Genes;2023-06-20

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