NRF2 Induction Supporting Breast Cancer Cell Survival Is Enabled by Oxidative Stress–Induced DPP3–KEAP1 Interaction-Reference-Cited by-同舟云学术

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NRF2 Induction Supporting Breast Cancer Cell Survival Is Enabled by Oxidative Stress–Induced DPP3–KEAP1 Interaction

Published:2017-04-17 Issue:11 Volume:77 Page:2881-2892
ISSN:0008-5472
Container-title:Cancer Research
language:en
Short-container-title:Cancer Res

Author:

Lu Kevin,Alcivar Allen L.,Ma Jianglin,Foo Tzeh Keong,Zywea Susan,Mahdi Amar,Huo Yanying,Kensler Thomas W.,Gatza Michael L.,Xia Bing

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 143 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Tumor suppressor KEAP1 promotes HSPA9 degradation, controlling mitochondrial biogenesis in breast cancer;Cell Reports;2024-07

2. Nuclear factor Erythroid 2-related factor (Nrf2): A potential therapeutic target for oral cancer - Molecular insights;Oral Oncology Reports;2024-06

3. Carbamate compounds induced toxic effects by affecting Nrf2 signaling pathways;Toxicology Reports;2024-06

4. DPP3 promotes breast cancer tumorigenesis by stabilizing FASN and promoting lipid synthesis;Acta Biochimica et Biophysica Sinica;2024-04-24

5. The Potential Role of Major Argan Oil Compounds as Nrf2 Regulators and Their Antioxidant Effects;Antioxidants;2024-03-13

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