GATM-Mediated Creatine Biosynthesis Enables Maintenance of FLT3-ITD–Mutant Acute Myeloid Leukemia
Author:
Funder
UF | UF Health | College of Medicine, University of Florida
Publisher
American Association for Cancer Research (AACR)
Subject
Cancer Research,Oncology,Molecular Biology
Cited by 6 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Evidence of an intracellular creatine-sensing mechanism that modulates creatine biosynthesis via AGAT expression in human HAP1 cells;Scientific Reports;2023-12-16
2. Suppression of the KRAS-NRF2 axis shifts arginine into the phosphocreatine energy system in pancreatic cancer cells;iScience;2023-12
3. A combinatorial therapeutic approach to enhance FLT3-ITD AML treatment;Cell Reports Medicine;2023-11
4. Investigation of the creatine biosynthesis negative feedback loop via creatine mediated control of arginine:glycine amidinotransferase expression in human HAP1 cells implies an intracellular creatine sensing mechanism;2023-09-08
5. Targeting OXPHOS de novo purine synthesis as the nexus of FLT3 inhibitor–mediated synergistic antileukemic actions;Science Advances;2022-09-16
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