FGFR1 Amplification Mediates Endocrine Resistance but Retains TORC Sensitivity in Metastatic Hormone Receptor–Positive (HR+) Breast Cancer-Reference-Cited by-同舟云学术

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FGFR1 Amplification Mediates Endocrine Resistance but Retains TORC Sensitivity in Metastatic Hormone Receptor–Positive (HR+) Breast Cancer

Published:2019-08-01 Issue:21 Volume:25 Page:6443-6451
ISSN:1078-0432
Container-title:Clinical Cancer Research
language:en
Short-container-title:Clin Cancer Res

Author:

Drago Joshua Z.,Formisano Luigi,Juric Dejan,Niemierko Andrzej,Servetto Alberto,Wander Seth A.,Spring Laura M.,Vidula Neelima,Younger Jerry,Peppercorn Jeffrey,Yuen Megan,Malvarosa Giuliana,Sgroi Dennis,Isakoff Steven J.,Moy Beverly,Ellisen Leif W.,Iafrate A. John,Arteaga Carlos L.,Bardia Aditya

Funder

Susan G Komen

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 60 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Design, synthesis and biological evaluation of 5-amino-1H-pyrazole-4-carboxamide derivatives as pan-FGFR covalent inhibitors;European Journal of Medicinal Chemistry;2024-09

2. Precision therapeutics and emerging strategies for HR-positive metastatic breast cancer;Nature Reviews Clinical Oncology;2024-08-23

3. Fibroblast growth factor receptor signaling in estrogen receptor-positive breast cancer: mechanisms and role in endocrine resistance;Frontiers in Oncology;2024-07-08

4. Overexpression of COL11A1 confers tamoxifen resistance in breast cancer;npj Breast Cancer;2024-05-28

5. FGFR4-driven plasticity in breast cancer progression and resistance to therapy;British Journal of Cancer;2024-04-17

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