Combined Inhibition of Janus Kinase 1/2 for the Treatment of JAK2V617F-Driven Neoplasms: Selective Effects on Mutant Cells and Improvements in Measures of Disease Severity-Reference-Cited by-同舟云学术

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Combined Inhibition of Janus Kinase 1/2 for the Treatment of JAK2V617F-Driven Neoplasms: Selective Effects on Mutant Cells and Improvements in Measures of Disease Severity

Published:2009-11-03 Issue:22 Volume:15 Page:6891-6900
ISSN:1078-0432
Container-title:Clinical Cancer Research
language:en
Short-container-title:Clin Cancer Res

Author:

Liu Phillip C.C.,Caulder Eian,Li Jun,Waeltz Paul,Margulis Alex,Wynn Richard,Becker-Pasha Mary,Li Yanlong,Crowgey Erin,Hollis Gregory,Haley Patrick,Sparks Richard B.,Combs Andrew P.,Rodgers James D.,Burn Timothy C.,Vaddi Kris,Fridman Jordan S.

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 17 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Cellular thermal shift assay (CETSA) for determining the drug binding affinity using Ba/F3 clones stably expressing receptor pseudokinases;Methods in Enzymology;2022

2. Primary myelofibrosis: Older age and high JAK2 V617F allele burden are associated with elevated plasma high-sensitivity C-reactive protein levels and a phenotype of progressive disease;Leukemia Research;2017-09

3. Targeting Upstream Janus Kinases;Cancer Drug Discovery and Development;2016

4. Co‐targeting the PI3K/ mTOR and JAK2 signalling pathways produces synergistic activity against myeloproliferative neoplasms;Journal of Cellular and Molecular Medicine;2013-11

5. Design and evaluation of novel 8-oxo-pyridopyrimidine Jak1/2 inhibitors;Bioorganic & Medicinal Chemistry Letters;2013-11

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