The Cytoskeleton Regulatory Protein hMena (ENAH) Is Overexpressed in Human Benign Breast Lesions with High Risk of Transformation and Human Epidermal Growth Factor Receptor-2–Positive/Hormonal Receptor

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The Cytoskeleton Regulatory Protein hMena (ENAH) Is Overexpressed in Human Benign Breast Lesions with High Risk of Transformation and Human Epidermal Growth Factor Receptor-2–Positive/Hormonal Receptor–Negative Tumors

Published:2006-03-01 Issue:5 Volume:12 Page:1470-1478
ISSN:1078-0432
Container-title:Clinical Cancer Research
language:en
Short-container-title:Clin Cancer Res

Author:

Di Modugno Francesca,Mottolese Marcella,Di Benedetto Anna,Conidi Andrea,Novelli Flavia,Perracchio Letizia,Venturo Irene,Botti Claudio,Jager Elke,Santoni Angela,Natali Pier Giorgio,Nisticò Paola

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 74 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. TRIM67 interacts with ENAH to regulate the apoptosis and autophagy of lung cancer cells;General physiology and biophysics;2024

2. Mena as a key enhancer factor of EMT to promote metastasis of human tongue squamous cell carcinoma;Oral Diseases;2023-05-19

3. DJ-1 promotes cell migration by interacting with Mena, the mammalian homolog of Drosophila enabled;Advances in Biological Regulation;2023-05

4. Matrix stiffness regulates tumor cell intravasation through expression and ESRP1-mediated alternative splicing of MENA;Cell Reports;2023-04

5. Mena regulates nesprin-2 to control actin–nuclear lamina associations, trans-nuclear membrane signalling and gene expression;Nature Communications;2023-03-23

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