Ablation of B7-H3 but Not B7-H4 Results in Highly Increased Tumor Burden in a Murine Model of Spontaneous Prostate Cancer-Reference-Cited by-同舟云学术

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Ablation of B7-H3 but Not B7-H4 Results in Highly Increased Tumor Burden in a Murine Model of Spontaneous Prostate Cancer

Published:2015-06-29 Issue:8 Volume:3 Page:849-854
ISSN:2326-6066
Container-title:Cancer Immunology Research
language:en
Short-container-title:Cancer Immunol Res

Author:

Kreymborg Katharina,Haak Stefan,Murali Rajmohan,Wei Joyce,Waitz Rebecca,Gasteiger Georg,Savage Peter A.,van den Brink Marcel R. M.,Allison James P.

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Immunology

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2. Dimerization of the 4Ig isoform of B7-H3 in tumor cells mediates enhanced proliferation and tumorigenic signaling;Communications Biology;2024-01-05

3. Immune checkpoint B7-H3 is a therapeutic vulnerability in prostate cancer harboring PTEN and TP53 deficiencies;Science Translational Medicine;2023-05-10

4. B7-H3 as a Therapeutic Target in Advanced Prostate Cancer;European Urology;2023-03

5. Inhibition of the B7-H3 immune checkpoint limits hepatocellular carcinoma progression by enhancing T lymphocyte-mediated immune cytotoxicity in vitro and in vivo;Clinical and Translational Oncology;2022-12-13

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