Differences in Expression Patterns of Cell Cycle Regulators after Cessation of Genotoxic and Non-genotoxic Carcinogen Treatment in the Rat Forestomach
Author:
Affiliation:
1. Department of Experimental Pathology and Tumor Biology Nagoya City University Graduate School of Medical Sciences
2. Food Safety Commission, Cabinet Office Government of Japan
Publisher
Japanese Society of Toxicologic Pathology
Subject
Toxicology,Pathology and Forensic Medicine
Link
http://www.jstage.jst.go.jp/article/tox/21/2/21_2_77/_pdf
Reference34 articles.
1. Comparison of Reversibility of Rat Forestomach Lesions Induced by Genotoxic and Non-genotoxic Carcinogens
2. Reversibility of Carcinogen-induced Rat Forestomach Basal Cell Hyperplasia Is Due to Squamous Cell Differentiation
3. 4. Ward JM. Dose response to a single injection of azoxymethane in rats. Induction of tumors in the gastrointestinal tract, auditory sebaceous glands, kidney, liver and preputial gland. Vet Pathol. 12: 165-177. 1975.
4. 5. Shirai T, Fukushima S, Ohshima M, Masuda A, and Ito N. Effects of butylated hydroxyanisole, butylated hydroxytoluene, and NaCl on gastric carcinogenesis initiated with N-methyl-N'-nitro-N-nitrosoguanidine in F344 rats. J Natl Cancer Inst. 72: 1189-1198. 1984.
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