Spontaneous hyaline glomerulopathy in a young Slc:ICR mouse
Author:
Affiliation:
1. Safety Research Laboratories, Sohyaku. Innovative Research Division, Mitsubishi Tanabe Pharma Corporation, 2-26-1 Muraoka-Higashi, Fujisawa, Kanagawa 251-8555, Japan
Publisher
Japanese Society of Toxicologic Pathology
Subject
Toxicology,Pathology and Forensic Medicine
Link
https://www.jstage.jst.go.jp/article/tox/33/4/33_2020-0027/_pdf
Reference18 articles.
1. 1. Homburger F, Russfield AB, Weisburger JH, Lim S, Chak SP, and Weisburger EK. Aging changes in CD-1 HaM/ICR mice reared under standard laboratory conditions. J Natl Cancer Inst. 55: 37–45. 1975.
2. 2. Yamate J, Tajima M, and Kudow S. Comparison of age-related changes between long-lived CRJ:CD-1 (ICR) and CRJ:B6C3F1 mice. Nippon Juigaku Zasshi. 48: 273–284. 1986.
3. 3. Ogura A, Asano T, Matsuda J, Takano K, Nakagawa M, and Fukui M. Characteristics of mutant mice (ICGN) with spontaneous renal lesions: a new model for human nephrotic syndrome. Lab Anim. 23: 169–174. 1989.
4. 4. Shibuya K, Tajima M, Yamate J, and Kudow S. Idiopathic glomerulopathy in a young ICR mouse. J Comp Pathol. 103: 107–111. 1990.
5. 5. Ago K, Sugahara G, Shirota K, and Kurata Y. Spontaneous early-onset glomerulonephritis in a 8-week-old male Crj:CD1 (ICR) mouse. J Toxicol Pathol. 28: 237–241. 2015.
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