Tracheal Morphologic and Protein Alterations FollowingShort-Term Cigarette Mainstream Smoke Exposure to Rats
Author:
Affiliation:
1. A. W. Spears Research Center, 420 N. English Street, Lorillard Tobacco Company, Greensboro, NC 27405, USA
2. EPL, Inc., Research Triangle Park, NC 27709, USA
Publisher
Japanese Society of Toxicologic Pathology
Subject
Toxicology,Pathology and Forensic Medicine
Link
https://www.jstage.jst.go.jp/article/tox/25/3/25_2012-0014/_pdf
Reference28 articles.
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2. 2. Carter CA, Doherty MM, Rusnak DW, Nettesheim P, and Ferriola PC. Alterations in the localization of F-actin, fibronectin, and thrombospondin occur prior to neoplastic transformation in rat tracheal epithelial cells. Exp Cell Res. 212: 141–150. 1994.
3. 3. Newland N, Baxter A, Hewitt K, and Minet E. CYP1A1/1B1 and CYP2A6/2A13 activity is conserved in cultures of differentiated primary human tracheobronchial epithelial cells. Toxicol In Vitro. 25: 922–929. 2011.
4. 4. Turetz ML, O’Connor TP, Tilley AE, Strulovici-Barel Y, Salit J, Dang D, Teater M, Mezey J, Clark AG, and Crystal RG. Trachea epithelium as a “canary” for cigarette smoking-induced biologic phenotype of the small airway epithelium. Clin Transl Sci. 2: 260–272. 2009.
5. 5. Battershill JM. The multiple chemicals and actions model of carcinogenesis. A possible new approach to developing prevention strategies for environmental carcinogenesis. Hum Exp Toxicol. 24: 547–558. 2005.
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