Analysis of Gene Expression Profile on Uterine Tumorigenesis Initiated with N-ethyl-N-nitrosourea and Inhibited by Ethinylestradiol in rasH2 Mice
Author:
Affiliation:
1. Laboratory of Veterinary Pathology, Tokyo University of Agriculture and Technology
2. Environmental Health Science Laboratory, Sumitomo Chemical Co., Ltd
3. Corporate Planning & Coordination Office, Sumitomo Chemical Co., Ltd
Publisher
Japanese Society of Toxicologic Pathology
Subject
Toxicology,Pathology and Forensic Medicine
Link
http://www.jstage.jst.go.jp/article/tox/17/3/17_3_155/_pdf
Reference30 articles.
1. Promoting effects of ethinyl estradiol on development of renal proliferative lesions induced byN-nitrosobis (2-oxopropyl)amine in female Syrian golden hamsters
2. Modifying Effects of Ethinylestradiol but Not Methoxychlor on N-Ethyl-N-nitrosourea-Induced Uterine Carcinogenesis in Heterozygous p53-Deficient CBA Mice
3. Estrogen receptor, epidermal growth factor receptor and cellular ploidy in elutriated subpopulations of hepatocytes during liver tumor promotion by 17α-ethinylestradiol in rats
4. Rapid induction of uterine endometrial proliferative lesions in transgenic mice carrying a human prototype c-Ha-ras gene (rasH2 mice) given a single intraperitoneal injection of N-ethyl-N-nitrosourea
5. Promoting Effects of Ethinylestradiol but not Atrazine on N-ethyl-N-nitrosourea-induced Uterine Carcinogenesis in ICR Mice
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1. Survey of tumorigenic sensitivity in 6-month rasH2-Tg mice studies compared with 2-year rodent assays;Journal of Toxicologic Pathology;2022
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