Chronic rapamycin treatment or lack ofS6K1does not reduce ribosome activity in vivo-Reference-Cited by-同舟云学术

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Chronic rapamycin treatment or lack ofS6K1does not reduce ribosome activity in vivo

Published:2013-08 Issue:15 Volume:12 Page:2493-2504
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Garelick Michael G,MacKay Vivian L,Yanagida Aya,Academia Emmeline C,Schreiber Katherine H,Ladiges Warren C,Kennedy Brian K

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

http://www.tandfonline.com/doi/pdf/10.4161/cc.25512

Reference62 articles.

1. mTOR is a key modulator of ageing and age-related disease

2. An anti-aging drug today: from senescence-promoting genes to anti-aging pill

3. Rapamycin Reverses Elevated mTORC1 Signaling in Lamin A/C–Deficient Mice, Rescues Cardiac and Skeletal Muscle Function, and Extends Survival

4. Ras, PI(3)K and mTOR signalling controls tumour cell growth

5. Rapamycin Extends Maximal Lifespan in Cancer-Prone Mice

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1. Inhibition of S6K lowers age-related inflammation and increases lifespan through the endolysosomal system;Nature Aging;2024-02-27

2. Stabilizing Immature Dendritic Spines in the Auditory Cortex: A Key Mechanism for mTORC1-Mediated Enhancement of Long-Term Fear Memories;The Journal of Neuroscience;2023-10-19

3. Mechanistic Target of Rapamycin Complex 1: From a Nutrient Sensor to a Key Regulator of Metabolism and Health;Advances in Nutrition;2022-09

4. Inhibition of S6K lowers age-related inflammation and immunosenescence and increases lifespan through the endolysosomal system;2022-08-26

5. The Translational Regulation in mTOR Pathway;Biomolecules;2022-06-08

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