JAK2-V617F-induced MAPK activity is regulated by PI3K and acts synergistically with PI3K on the proliferation of JAK2-V617F-positive cells-Reference-Cited by-同舟云学术

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JAK2-V617F-induced MAPK activity is regulated by PI3K and acts synergistically with PI3K on the proliferation of JAK2-V617F-positive cells

Published:2013-07-15 Issue:3 Volume:2 Page:e24574
ISSN:2162-3996
Container-title:JAK-STAT
language:en
Short-container-title:JAK-STAT

Author:

Wolf Alexandra,Eulenfeld René,Gäbler Karoline,Rolvering Catherine,Haan Serge,Behrmann Iris,Denecke Bernd,Haan Claude,Schaper Fred

Publisher

Informa UK Limited

Subject

Cancer Research,Cell Biology,Molecular Biology

Link

http://www.tandfonline.com/doi/pdf/10.1080/10256018808623883

Reference65 articles.

1. Acquiring signalling specificity from the cytokine receptor gp130

2. Widespread occurrence of the JAK2 V617F mutation in chronic myeloproliferative disorders

3. A Gain-of-Function Mutation ofJAK2in Myeloproliferative Disorders

4. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders

5. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera

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1. MAPK-negative feedback regulation confers dependence to JAK2V617F signaling;Leukemia;2023-07-10

2. EBF1–JAK2 inhibits the PAX5 function through physical interaction with PAX5 and kinase activity;International Journal of Hematology;2023-05-07

3. PI3K Inhibition Restores and Amplifies Response to Ruxolitinib in Patients with Myelofibrosis;Clinical Cancer Research;2023-04-10

4. The implications of alternative pre-mRNA splicing in cell signal transduction;Experimental & Molecular Medicine;2023-04-03

5. Increase in Frequency of Myeloid-Derived Suppressor Cells in the Bone Marrow of Myeloproliferative Neoplasm: Potential Implications in Myelofibrosis;Advances in Experimental Medicine and Biology;2023

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